Tuesday, July 28, 2009

FROM THE ARCHIVE

Recirculation of researchers with Chronic Fellow Syndrome is highly dependent on self-recognition at homing compartments

J.C., Jr
MPI, Munich, Germany

The pathology of Chronic Fellow Syndrome (CFS) leads to young scientists working at foreign countries to a non-saturable, stationary state, in the progress of their professional achievements, which has been correlated with their incapability to maintain the proper growing-rate of their curriculum vitae after the first, naive years, of their research career. Epidemiological and histopathological post-mortem studies have shown that CFS etiological agent is brain specific, since foci of infection are exclusively found in the minds of the affected individuals. Triggering of CFS is dependent on both environmental and godfather-inherited factors, and its development has been suggested to be associated with the acquisition of related disorders, such as Friend-Sick Syndrome. In addition, previous reports have pointed out the importance of committed overseas recirculation suffered by patients with CFS in the aggravation of the disease (see CurrRevol 08/06/09), which may ultimately direct them into an irreversible erratic stage of gypsy-like professional behaviour, loss of friendship-related feelings, early-morning acute laziness and, eventually, the big sleep. In an attempt to understand at the topological level the routes of progression of the syndrome, we have analyzed the fine specificity in the trafficking of a CFS patient committed to recirculate at home country institutions. To this end, the possibility has been tested of bypassing the previously acquired commitment through an alternative allocation pathway which involved a fellowship application at a neighbour country that, otherwise, showed no cultural direct bonds with the patient´s native land. To avoid results which could be due to a lack of professional merits in the applicant, the patient was primed with a boosting dose of high quality publications just before the application process was carried out. As a recipient of such application, a very reliable European research organisation was chosen. As expected, on the basis of a standard foreign agent rejection response, this organisation could not award the fellowship to the patient. Interestingly, in situ analysis of the future prospects of the patient`s non-self new allocation, revealed a remarkable short-term involution of such compartment, which will likely be followed by homeostatic reabsorption or the essential self-materials, wall decay, and vanishing. Thus, although alternative pathways of allocation can momentarily divert fellows with CFS through non-self locations, our results indicate that self-recognition at homing compartments is necessary for the aberrant recirculation of researchers with CFS.

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